Passiv rygning er en risikofaktor for udvikling af nedsat lungefunktion og KOL

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Standard

Passiv rygning er en risikofaktor for udvikling af nedsat lungefunktion og KOL. / Robertson, Samuel; Pisinger, Charlotta H; Løkke, Anders; Godtfredsen, Nina S.

I: Ugeskrift for Laeger, Bind 181, Nr. 8, V08180584, 2019.

Publikation: Bidrag til tidsskriftTidsskriftartikelfagfællebedømt

Harvard

Robertson, S, Pisinger, CH, Løkke, A & Godtfredsen, NS 2019, 'Passiv rygning er en risikofaktor for udvikling af nedsat lungefunktion og KOL', Ugeskrift for Laeger, bind 181, nr. 8, V08180584. <https://ugeskriftet.dk/videnskab/passiv-rygning-er-en-risikofaktor-udvikling-af-nedsat-lungefunktion-og-kol>

APA

Robertson, S., Pisinger, C. H., Løkke, A., & Godtfredsen, N. S. (2019). Passiv rygning er en risikofaktor for udvikling af nedsat lungefunktion og KOL. Ugeskrift for Laeger, 181(8), [V08180584]. https://ugeskriftet.dk/videnskab/passiv-rygning-er-en-risikofaktor-udvikling-af-nedsat-lungefunktion-og-kol

Vancouver

Robertson S, Pisinger CH, Løkke A, Godtfredsen NS. Passiv rygning er en risikofaktor for udvikling af nedsat lungefunktion og KOL. Ugeskrift for Laeger. 2019;181(8). V08180584.

Author

Robertson, Samuel ; Pisinger, Charlotta H ; Løkke, Anders ; Godtfredsen, Nina S. / Passiv rygning er en risikofaktor for udvikling af nedsat lungefunktion og KOL. I: Ugeskrift for Laeger. 2019 ; Bind 181, Nr. 8.

Bibtex

@article{998d8563b4484627ae84d21fb4ede521,
title = "Passiv rygning er en risikofaktor for udvikling af nedsat lungefunktion og KOL",
abstract = "The purpose of this review is to summarise the existing knowledge of the relationship between exposure to en-vironmental tobacco smoke (ETS) and the development of chronic obstructive pulmonary disease (COPD). A possible causal relationship between ETS and COPD has long been suspected. Prenatal as well as postnatal ETS can reduce the lung function of the child permanently. Albeit the causality is multifactorial, new evidence has emerged since the Surgeon General's report from 2006 and suggests, that ETS is an independent and significant risk factor for developing COPD.",
keywords = "Child, Female, Humans, Pregnancy, Pulmonary Disease, Chronic Obstructive/epidemiology, Risk Factors, Tobacco Smoke Pollution",
author = "Samuel Robertson and Pisinger, {Charlotta H} and Anders L{\o}kke and Godtfredsen, {Nina S}",
year = "2019",
language = "Dansk",
volume = "181",
journal = "Ugeskrift for Laeger",
issn = "0041-5782",
publisher = "Almindelige Danske Laegeforening",
number = "8",

}

RIS

TY - JOUR

T1 - Passiv rygning er en risikofaktor for udvikling af nedsat lungefunktion og KOL

AU - Robertson, Samuel

AU - Pisinger, Charlotta H

AU - Løkke, Anders

AU - Godtfredsen, Nina S

PY - 2019

Y1 - 2019

N2 - The purpose of this review is to summarise the existing knowledge of the relationship between exposure to en-vironmental tobacco smoke (ETS) and the development of chronic obstructive pulmonary disease (COPD). A possible causal relationship between ETS and COPD has long been suspected. Prenatal as well as postnatal ETS can reduce the lung function of the child permanently. Albeit the causality is multifactorial, new evidence has emerged since the Surgeon General's report from 2006 and suggests, that ETS is an independent and significant risk factor for developing COPD.

AB - The purpose of this review is to summarise the existing knowledge of the relationship between exposure to en-vironmental tobacco smoke (ETS) and the development of chronic obstructive pulmonary disease (COPD). A possible causal relationship between ETS and COPD has long been suspected. Prenatal as well as postnatal ETS can reduce the lung function of the child permanently. Albeit the causality is multifactorial, new evidence has emerged since the Surgeon General's report from 2006 and suggests, that ETS is an independent and significant risk factor for developing COPD.

KW - Child

KW - Female

KW - Humans

KW - Pregnancy

KW - Pulmonary Disease, Chronic Obstructive/epidemiology

KW - Risk Factors

KW - Tobacco Smoke Pollution

M3 - Tidsskriftartikel

C2 - 30821243

VL - 181

JO - Ugeskrift for Laeger

JF - Ugeskrift for Laeger

SN - 0041-5782

IS - 8

M1 - V08180584

ER -

ID: 235858780